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    ltered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highes

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    Antioxidants have been widely praised in the media. Many know they are linked with anti-aging properties. However this is not new, it all began with one man’s theory a half a century ago about how free radicals were associated with aging, and science has been trying to catch up with it ever since.

    As Dr. Nicholas Perricone, M.D., states in his 2001 book, “The Wrinkle Cure”: “When it comes to aging, it’s not Father Time that’s public enemy number 1. It’s the very busy, very nasty little molecule called the free radical.”

    The Free Radical Theory of Aging was published by Denman Harman in 1956. He theorized that aging is a result of free radical damage of the cells of the body. This is also called oxidative stress.

    Today, a great deal of experimental evidence supports the premise that length of life is determined by the crucial balance of antioxidants with free radicals in the body. Oxidative stress is being shown to be at the root of disease and aging.

    One example is that the life of the fruit fly was up to 30% longer when it was genetically altered with an addition of enzymatic antioxidants. Not only that, but the altered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highes

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    er since.

    As Dr. Nicholas Perricone, M.D., states in his 2001 book, “The Wrinkle Cure”: “When it comes to aging, it’s not Father Time that’s public enemy number 1. It’s the very busy, very nasty little molecule called the free radical.”

    The Free Radical Theory of Aging was published by Denman Harman in 1956. He theorized that aging is a result of free radical damage of the cells of the body. This is also called oxidative stress.

    Today, a great deal of experimental evidence supports the premise that length of life is determined by the crucial balance of antioxidants with free radicals in the body. Oxidative stress is being shown to be at the root of disease and aging.

    One example is that the life of the fruit fly was up to 30% longer when it was genetically altered with an addition of enzymatic antioxidants. Not only that, but the altered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highes

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    lished by Denman Harman in 1956. He theorized that aging is a result of free radical damage of the cells of the body. This is also called oxidative stress.

    Today, a great deal of experimental evidence supports the premise that length of life is determined by the crucial balance of antioxidants with free radicals in the body. Oxidative stress is being shown to be at the root of disease and aging.

    One example is that the life of the fruit fly was up to 30% longer when it was genetically altered with an addition of enzymatic antioxidants. Not only that, but the altered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highes

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    ntioxidants with free radicals in the body. Oxidative stress is being shown to be at the root of disease and aging.

    One example is that the life of the fruit fly was up to 30% longer when it was genetically altered with an addition of enzymatic antioxidants. Not only that, but the altered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highes

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    ltered fruit flies also showed a reduced amount of age-related oxidative damage.

    Studies of humans have also shown evidence of free radical damage playing a large part in human aging. One 1996 study compared markers of free radical damage in the blood and found evidence of the highest oxidative damage associated with the disabled elderly, an intermediate amount with the healthy elderly, and the lowest levels with the healthy adults.

    The study also found that higher blood levels of antioxidant Vitamins C and E were associated with less disability, and signs of free radical damage were associated with more disability.

    We need to rethink our concept of aging. We accept disease, disability, senility, wrinkles, and all the other many signs of aging as natural. Instead, we should view this as ‘unsuccessful aging’ – ie., aging associated with deterioration, disease and disability.

    Successful aging is what happens when the human body is able to fight off oxidative stress, and continue to regenerate and repair itself. Successful aging is getting older healthily, without significant pathological conditions.

    What the evidence is telling us is that it is crucial we take antioxidants and free radicals very seriously if we want to ‘age gracefully’ and avoid the many pitfalls of ‘unsuccessful aging’.

    Even young people can be victims of unsuccessful aging, if they are not providing their bodies with the nece

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